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64 channel linear silicon probes  (NeuroNexus Technologies)


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    Structured Review

    NeuroNexus Technologies 64 channel linear silicon probes
    64 Channel Linear Silicon Probes, supplied by NeuroNexus Technologies, used in various techniques. Bioz Stars score: 97/100, based on 2500 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/64+channel+linear+silicon+probes/Silicon+Neural+Probe+%2F+Silicon+Microelectrode+Array/pm40499534-321-9-13
    Average 97 stars, based on 2500 article reviews
    64 channel linear silicon probes - by Bioz Stars, 2026-09
    97/100 stars

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    Related Articles

    other:

    Article Title: Hippocampal 4-Hz oscillations emerge during stationary running in a wheel and are resistant to medial septum inactivation.
    Article Snippet: Two 64-channel linear silicon probes (Neuronexus or Janelia RC) were bilaterally implanted at the dorsal CA1 area (coordinates: -4.0 mm AP, ± 3 mm ML) of the rat hippocampus (n = 3 animals across ten sessions).

    Activity Assay:

    Article Title: Adaptive reinforcement learning is causally supported by anterior cingulate cortex and striatum.
    Article Snippet: Article Adaptive reinforcement learning is causally supported by anterior cingulate cortex and striatum Graphical abstract Highlights • Gaze-contingent stimulation changes learning of feature values at high uncertainty • ACC simulation impairs weighting of error histories and optimizing exploration • Striatum simulation improves use of prediction errors to update value expectations • Stimulation induces uncertainty in ACC but in striatum augments outcome certainty Authors Robert Louis Treuting, Kianoush Banaie Boroujeni, Charles Grimes Gerrity, Adam Neumann, Paul Tiesinga, Thilo Womelsdorf Correspondence thilo.womelsdorf@vanderbilt.edu In brief Treuting et al. show that microstimulation in ACC and striatum causally affects the learning and updating of feature-based attention.. ACC stimulation impairs exploring relevant objects, which is linked to increased uncertainty about previous outcomes.. Striatum stimulation improves exploring relevant objects, which is linked to a more efficient updating of relevant feature values.



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    a) Left: schematic of CreOn/FlpOn-ChR2-EYFP injections. Right: example ChR2 expression patterns (scale: 200µm). b) Schematic of <t>linear</t> <t>silicon</t> <t>probe</t> attached to a tapered optical fiber with example LFPs and units shown in their respective layer locations. c) Stimulation protocol schematic showing spontaneous and optogenetic stimulation (opto) blocks. Opto blocks include periods of stimulation and intertrial-intervals (ITIs). d) Left: Mean LFP power change across tissue depth. Center: change in delta power (1-4Hz) during slow wave sleep (SWS) across cortical layers. Right: delta band power change across SWS, quiet wake (QW) and movement (Move) (p = 0.003, 0.008, 0.069, resp., paired t-test). e) Normalized cross-correlograms (CCGs) between stimulated periods and ITIs. Left: mean CCG. Center: distribution of peak synchrony changes for each pair. Right: mean ± s.e.m. of pairwise synchrony changes (p < 0.001, paired t-test). f) Change in pairwise synchrony across SWS, QW, and Move (p < 0.001 for all, paired t-test). g) Spike-phase coherence across the LFP frequency spectrum during stimulation and spontaneous blocks. h) Distribution (left) and average (right) of unit-wise change in delta band phase locking with stimulation (p < 0.001, paired t-test). i) Example LFP data and single unit activity during UP and DOWN state transitions during spontaneous blocks. j) Average LFP time-locked to the peak of the DOWN state with (blue) and without (grey) optogenetic stimulation in example session. k) Change in amplitude and duration of DOWN state with optogenetic stimulation (p = 0.021 and p = 0.003, respectively, paired t-test). N = 8 sessions across 4 mice, N = 446 units and 39,804 pairs. ***: p < 0.001, **: p < 0.01, *: p < 0.05; ns, not significant. Data are means, shading and bars indicate s.e.m.
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    a) Left: schematic of CreOn/FlpOn-ChR2-EYFP injections. Right: example ChR2 expression patterns (scale: 200µm). b) Schematic of <t>linear</t> <t>silicon</t> <t>probe</t> attached to a tapered optical fiber with example LFPs and units shown in their respective layer locations. c) Stimulation protocol schematic showing spontaneous and optogenetic stimulation (opto) blocks. Opto blocks include periods of stimulation and intertrial-intervals (ITIs). d) Left: Mean LFP power change across tissue depth. Center: change in delta power (1-4Hz) during slow wave sleep (SWS) across cortical layers. Right: delta band power change across SWS, quiet wake (QW) and movement (Move) (p = 0.003, 0.008, 0.069, resp., paired t-test). e) Normalized cross-correlograms (CCGs) between stimulated periods and ITIs. Left: mean CCG. Center: distribution of peak synchrony changes for each pair. Right: mean ± s.e.m. of pairwise synchrony changes (p < 0.001, paired t-test). f) Change in pairwise synchrony across SWS, QW, and Move (p < 0.001 for all, paired t-test). g) Spike-phase coherence across the LFP frequency spectrum during stimulation and spontaneous blocks. h) Distribution (left) and average (right) of unit-wise change in delta band phase locking with stimulation (p < 0.001, paired t-test). i) Example LFP data and single unit activity during UP and DOWN state transitions during spontaneous blocks. j) Average LFP time-locked to the peak of the DOWN state with (blue) and without (grey) optogenetic stimulation in example session. k) Change in amplitude and duration of DOWN state with optogenetic stimulation (p = 0.021 and p = 0.003, respectively, paired t-test). N = 8 sessions across 4 mice, N = 446 units and 39,804 pairs. ***: p < 0.001, **: p < 0.01, *: p < 0.05; ns, not significant. Data are means, shading and bars indicate s.e.m.
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    Image Search Results


    a) Left: schematic of CreOn/FlpOn-ChR2-EYFP injections. Right: example ChR2 expression patterns (scale: 200µm). b) Schematic of linear silicon probe attached to a tapered optical fiber with example LFPs and units shown in their respective layer locations. c) Stimulation protocol schematic showing spontaneous and optogenetic stimulation (opto) blocks. Opto blocks include periods of stimulation and intertrial-intervals (ITIs). d) Left: Mean LFP power change across tissue depth. Center: change in delta power (1-4Hz) during slow wave sleep (SWS) across cortical layers. Right: delta band power change across SWS, quiet wake (QW) and movement (Move) (p = 0.003, 0.008, 0.069, resp., paired t-test). e) Normalized cross-correlograms (CCGs) between stimulated periods and ITIs. Left: mean CCG. Center: distribution of peak synchrony changes for each pair. Right: mean ± s.e.m. of pairwise synchrony changes (p < 0.001, paired t-test). f) Change in pairwise synchrony across SWS, QW, and Move (p < 0.001 for all, paired t-test). g) Spike-phase coherence across the LFP frequency spectrum during stimulation and spontaneous blocks. h) Distribution (left) and average (right) of unit-wise change in delta band phase locking with stimulation (p < 0.001, paired t-test). i) Example LFP data and single unit activity during UP and DOWN state transitions during spontaneous blocks. j) Average LFP time-locked to the peak of the DOWN state with (blue) and without (grey) optogenetic stimulation in example session. k) Change in amplitude and duration of DOWN state with optogenetic stimulation (p = 0.021 and p = 0.003, respectively, paired t-test). N = 8 sessions across 4 mice, N = 446 units and 39,804 pairs. ***: p < 0.001, **: p < 0.01, *: p < 0.05; ns, not significant. Data are means, shading and bars indicate s.e.m.

    Journal: bioRxiv

    Article Title: Neocortical long-range inhibition promotes cortical synchrony and sleep

    doi: 10.1101/2024.06.20.599756

    Figure Lengend Snippet: a) Left: schematic of CreOn/FlpOn-ChR2-EYFP injections. Right: example ChR2 expression patterns (scale: 200µm). b) Schematic of linear silicon probe attached to a tapered optical fiber with example LFPs and units shown in their respective layer locations. c) Stimulation protocol schematic showing spontaneous and optogenetic stimulation (opto) blocks. Opto blocks include periods of stimulation and intertrial-intervals (ITIs). d) Left: Mean LFP power change across tissue depth. Center: change in delta power (1-4Hz) during slow wave sleep (SWS) across cortical layers. Right: delta band power change across SWS, quiet wake (QW) and movement (Move) (p = 0.003, 0.008, 0.069, resp., paired t-test). e) Normalized cross-correlograms (CCGs) between stimulated periods and ITIs. Left: mean CCG. Center: distribution of peak synchrony changes for each pair. Right: mean ± s.e.m. of pairwise synchrony changes (p < 0.001, paired t-test). f) Change in pairwise synchrony across SWS, QW, and Move (p < 0.001 for all, paired t-test). g) Spike-phase coherence across the LFP frequency spectrum during stimulation and spontaneous blocks. h) Distribution (left) and average (right) of unit-wise change in delta band phase locking with stimulation (p < 0.001, paired t-test). i) Example LFP data and single unit activity during UP and DOWN state transitions during spontaneous blocks. j) Average LFP time-locked to the peak of the DOWN state with (blue) and without (grey) optogenetic stimulation in example session. k) Change in amplitude and duration of DOWN state with optogenetic stimulation (p = 0.021 and p = 0.003, respectively, paired t-test). N = 8 sessions across 4 mice, N = 446 units and 39,804 pairs. ***: p < 0.001, **: p < 0.01, *: p < 0.05; ns, not significant. Data are means, shading and bars indicate s.e.m.

    Article Snippet: A 64 channel linear silicon probe (H3 probe, Cambridge Neurotech) physically coupled to a tapered optical fiber was slowly (1μm/sec, over ∼20 minutes) inserted into V1.

    Techniques: Expressing, Activity Assay